The vast majority of low-grade B-cell lymphomas of the stomach are derived from mucosa-associated lymphoid tissue, and are therefore of the MALT type. Approximately 40 to 50 percent present with indolent or low-grade histology, of which 70 to 80 percent are confined to the stomach (stage IE) An increasing body of evidence has shown that the development of MALT lymphomas (also called MALTomas) is due to the clonal expansion of B-cells that accompanies chronic gastritis in the presence of Helicobacter pylori. Molecular techniques have become so sensitive at detecting early emergence of B-cell clonal expansion of gastric lymphocytes, hat the distinction between chronic gastritis and low-grade MALT lymphomas has blurred.
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